Rosemary J. Akhurst, PhD
Professor In Residence, Helen Diller Family Comprehensive Cancer Center and Department of Anatomy, UCSF; Director, Preclinical Therapeutics Core Facility, Helen Diller Family Comprehensive Cancer Center, UCSF
Professor In Residence, Helen Diller Family Comprehensive Cancer Center and Department of Anatomy, UCSF; Director, Preclinical Therapeutics Core Facility, Helen Diller Family Comprehensive Cancer Center, UCSF
As a UCSF Professor of Anatomy in the Helen Diller Family Comprehensive Cancer Center, my laboratory studies the roles and mechanism of action of TGFβ signaling in cancer, development, and vascular biology. We investigate chemically-induced carcinomas, namely DMBA/TPA-induced cutaneous squamous cell carcinomas that are genetically heterogenous and share many features with human cancers. Our studies were the first to show that TGFβ1 is induced by tumor promotion and that it can play both positive and negative roles in tumor initiation and progression in vivo. TGF has pleiotropic activity and we showed that this growth factor can drive epithelial-mesenchymal transformation of tumor cells and cancer stem cell maintenance. It is also a major regulator of the tumor microenvironment through its effects on vascular cells, angiogenesis, and as a potent immune-suppressor, all activities that drive tumor progression and metastasis.
Currently, we undertake translational studies investigating how targeting of distinct TGFβ signaling components can be used therapeutically, particularly to augment cancer immunotherapy, and our studies have contributed to the initiation of ongoing oncology clinical trials of novel anti-TGFβ signaling agents by Novartis and Pfizer. We are interested in the influence of genetic background on responses to cancer therapies. As such. we use mouse models to investigate how germline genetic variants influence responses to immunotherapies through effects on tumor immunity and the tumor microenvironment.
Finally, we have a deep interest in the cellular and molecular mechanisms involved in maintenance of vascular integrity, and how these go awry in disease states, such as in the vascular pathology of the human genetic disorder, Hereditary Hemorrhagic Telangiectasia (HHT). HHT results from germline loss of function mutations in genes encoding TGFβ/BMP signaling molecules, endoglin, ACVRL1/Alk1, or Smad4, and HHT disease progression is influenced by germline genetic variants and by environmental factors such as inflammation and wounding. Our studies often involve large collaborative teams.
As Faculty Director of the NCI-sponsored CCSG Preclinical Therapeutics Core (PTC), my interest is in providing access to state-of-the-art technology and technical support for small animal cancer therapeutics and imaging, for members of the HDFCCC scientific community and beyond. Through competitive federal and non-federal awards, we have acquired instrumentation for all three UCSF cancer campuses. These include Vevo770 and Vevo2100 ultrasound imaging platforms, several Perkin Elmer IVIS Spectrum multichannel fluorescent/ bioluminescent imagers, an Xstrahl small animal micro-CT-guided irradiator, and a Perkin Elmer, high-resolution high through put micro-CT scanner.
Imperial College of Science and Technology, University of London, UK, BSc (Hons), 1st class, 1975-1978, Biochemistry
Beatson Institute for Cancer Research, Glasgow, UK, PhD, 1978-1981, Molecular Biology