Transient receptor potential ion channels V4 and A1 contribute to pancreatitis pain in mice. Read more about Transient receptor potential ion channels V4 and A1 contribute to pancreatitis pain in mice.
Agonists of protease-activated receptors 1 and 2 stimulate electrolyte secretion from mouse gallbladder. Read more about Agonists of protease-activated receptors 1 and 2 stimulate electrolyte secretion from mouse gallbladder.
Altered inflammatory gene expression underlies increased susceptibility to murine postoperative ileus with advancing age. Read more about Altered inflammatory gene expression underlies increased susceptibility to murine postoperative ileus with advancing age.
Role of calcitonin receptor-like receptor in colonic motility and inflammation. Read more about Role of calcitonin receptor-like receptor in colonic motility and inflammation.
ANIT toxicity toward mouse hepatocytes in vivo is mediated primarily by neutrophils via CD18. Read more about ANIT toxicity toward mouse hepatocytes in vivo is mediated primarily by neutrophils via CD18.
Transient receptor potential vanilloid 1, calcitonin gene-related peptide, and substance P mediate nociception in acute pancreatitis. Read more about Transient receptor potential vanilloid 1, calcitonin gene-related peptide, and substance P mediate nociception in acute pancreatitis.
Limited role for CXC chemokines in the pathogenesis of alpha-naphthylisothiocyanate-induced liver injury. Read more about Limited role for CXC chemokines in the pathogenesis of alpha-naphthylisothiocyanate-induced liver injury.
Tyrphostin AG 126 inhibits development of postoperative ileus induced by surgical manipulation of murine colon. Read more about Tyrphostin AG 126 inhibits development of postoperative ileus induced by surgical manipulation of murine colon.
Expression and endocytosis of VEGF and its receptors in human colonic vascular endothelial cells. Read more about Expression and endocytosis of VEGF and its receptors in human colonic vascular endothelial cells.
Deletion of neutral endopeptidase exacerbates intestinal inflammation induced by Clostridium difficile toxin A. Read more about Deletion of neutral endopeptidase exacerbates intestinal inflammation induced by Clostridium difficile toxin A.
