Functional contributions of N- and O-glycans to L-selectin ligands in murine and human lymphoid organs. Read more about Functional contributions of N- and O-glycans to L-selectin ligands in murine and human lymphoid organs.
Angiopoietin-2-driven vascular remodeling in airway inflammation. Read more about Angiopoietin-2-driven vascular remodeling in airway inflammation.
Antibody treatment promotes compensation for human cytomegalovirus-induced pathogenesis and a hypoxia-like condition in placentas with congenital infection. Read more about Antibody treatment promotes compensation for human cytomegalovirus-induced pathogenesis and a hypoxia-like condition in placentas with congenital infection.
The DNA damage-binding protein XPC is a frequent target for inactivation in squamous cell carcinomas. Read more about The DNA damage-binding protein XPC is a frequent target for inactivation in squamous cell carcinomas.
Tissue slice grafts: an in vivo model of human prostate androgen signaling. Read more about Tissue slice grafts: an in vivo model of human prostate androgen signaling.
Ricinus communis agglutinin I leads to rapid down-regulation of VEGFR-2 and endothelial cell apoptosis in tumor blood vessels. Read more about Ricinus communis agglutinin I leads to rapid down-regulation of VEGFR-2 and endothelial cell apoptosis in tumor blood vessels.
Angiopoietin/Tie2 signaling transforms capillaries into venules primed for leukocyte trafficking in airway inflammation. Read more about Angiopoietin/Tie2 signaling transforms capillaries into venules primed for leukocyte trafficking in airway inflammation.
Steroid-resistant lymphatic remodeling in chronically inflamed mouse airways. Read more about Steroid-resistant lymphatic remodeling in chronically inflamed mouse airways.
Reduced expression of integrin alphavbeta8 is associated with brain arteriovenous malformation pathogenesis. Read more about Reduced expression of integrin alphavbeta8 is associated with brain arteriovenous malformation pathogenesis.
Trophoblastic neoplasms express fatty acid synthase, which may be a therapeutic target via its inhibitor C93. Read more about Trophoblastic neoplasms express fatty acid synthase, which may be a therapeutic target via its inhibitor C93.
