alpha beta T cell development is abolished in mice lacking both Lck and Fyn protein tyrosine kinases. Read more about alpha beta T cell development is abolished in mice lacking both Lck and Fyn protein tyrosine kinases.
CD28/B7 regulation of Th1 and Th2 subsets in the development of autoimmune diabetes. Read more about CD28/B7 regulation of Th1 and Th2 subsets in the development of autoimmune diabetes.
CD94 and a novel associated protein (94AP) form a NK cell receptor involved in the recognition of HLA-A, HLA-B, and HLA-C allotypes. Read more about CD94 and a novel associated protein (94AP) form a NK cell receptor involved in the recognition of HLA-A, HLA-B, and HLA-C allotypes.
DNAM-1, a novel adhesion molecule involved in the cytolytic function of T lymphocytes. Read more about DNAM-1, a novel adhesion molecule involved in the cytolytic function of T lymphocytes.
Vav and SLP-76 interact and functionally cooperate in IL-2 gene activation. Read more about Vav and SLP-76 interact and functionally cooperate in IL-2 gene activation.
Essential role for cathepsin S in MHC class II-associated invariant chain processing and peptide loading. Read more about Essential role for cathepsin S in MHC class II-associated invariant chain processing and peptide loading.
Assembly of the truncated immunoglobulin heavy chain D mu into antigen receptor-like complexes in pre-B cells but not in B cells. Read more about Assembly of the truncated immunoglobulin heavy chain D mu into antigen receptor-like complexes in pre-B cells but not in B cells.
Blockade of CD28/B7-1 interaction prevents epitope spreading and clinical relapses of murine EAE. Read more about Blockade of CD28/B7-1 interaction prevents epitope spreading and clinical relapses of murine EAE.
Antigen-induced exclusion from follicles and anergy are separate and complementary processes that influence peripheral B cell fate. Read more about Antigen-induced exclusion from follicles and anergy are separate and complementary processes that influence peripheral B cell fate.
Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4. Read more about Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4.
