Superenhancer activation of KLHDC8A drives glioma ciliation and hedgehog signaling. Read more about Superenhancer activation of KLHDC8A drives glioma ciliation and hedgehog signaling.
CBFA2T3-GLIS2 model of pediatric acute megakaryoblastic leukemia identifies FOLR1 as a CAR T cell target. Read more about CBFA2T3-GLIS2 model of pediatric acute megakaryoblastic leukemia identifies FOLR1 as a CAR T cell target.
Erythroid lineage Jak2V617F expression promotes atherosclerosis through erythrophagocytosis and macrophage ferroptosis. Read more about Erythroid lineage Jak2V617F expression promotes atherosclerosis through erythrophagocytosis and macrophage ferroptosis.
Breast cancer chemotherapy induces vascular dysfunction and hypertension through a NOX4-dependent mechanism. Read more about Breast cancer chemotherapy induces vascular dysfunction and hypertension through a NOX4-dependent mechanism.
Deficiency of the splicing factor RBM10 limits EGFR inhibitor response in EGFR-mutant lung cancer. Read more about Deficiency of the splicing factor RBM10 limits EGFR inhibitor response in EGFR-mutant lung cancer.
Mass cytometry detects H3.3K27M-specific vaccine responses in diffuse midline glioma. Read more about Mass cytometry detects H3.3K27M-specific vaccine responses in diffuse midline glioma.
Sepsis promotes splenic production of a protective platelet pool with high CD40 ligand expression. Read more about Sepsis promotes splenic production of a protective platelet pool with high CD40 ligand expression.
Metronidazole treatment rapidly reduces genital inflammation through effects on bacterial vaginosis-associated bacteria rather than lactobacilli. Read more about Metronidazole treatment rapidly reduces genital inflammation through effects on bacterial vaginosis-associated bacteria rather than lactobacilli.
Microbial signals, MyD88, and lymphotoxin drive TNF-independent intestinal epithelial tissue damage. Read more about Microbial signals, MyD88, and lymphotoxin drive TNF-independent intestinal epithelial tissue damage.
Remodeling of the tumor/tumor microenvironment ecosystem during KRAS G12C inhibitor clinical resistance in lung cancer. Read more about Remodeling of the tumor/tumor microenvironment ecosystem during KRAS G12C inhibitor clinical resistance in lung cancer.
