TIM-2 is expressed on B cells and in liver and kidney and is a receptor for H-ferritin endocytosis. Read more about TIM-2 is expressed on B cells and in liver and kidney and is a receptor for H-ferritin endocytosis.
Aberrant T cell differentiation in the absence of Dicer. Read more about Aberrant T cell differentiation in the absence of Dicer.
Granulocyte/macrophage colony-stimulating factor and accessory cells modulate radioprotection by purified hematopoietic cells. Read more about Granulocyte/macrophage colony-stimulating factor and accessory cells modulate radioprotection by purified hematopoietic cells.
Twisting tails exposed: the evidence for TCR conformational change. Read more about Twisting tails exposed: the evidence for TCR conformational change.
Cyclical modulation of sphingosine-1-phosphate receptor 1 surface expression during lymphocyte recirculation and relationship to lymphoid organ transit. Read more about Cyclical modulation of sphingosine-1-phosphate receptor 1 surface expression during lymphocyte recirculation and relationship to lymphoid organ transit.
The role of innate immunity in autoimmunity. Read more about The role of innate immunity in autoimmunity.
A Leishmania major response locus identified by interval-specific congenic mapping of a T helper type 2 cell bias-controlling quantitative trait locus. Read more about A Leishmania major response locus identified by interval-specific congenic mapping of a T helper type 2 cell bias-controlling quantitative trait locus.
Loss of synchronized retinal phagocytosis and age-related blindness in mice lacking alphavbeta5 integrin. Read more about Loss of synchronized retinal phagocytosis and age-related blindness in mice lacking alphavbeta5 integrin.
ADAP-SLP-76 binding differentially regulates supramolecular activation cluster (SMAC) formation relative to T cell-APC conjugation. Read more about ADAP-SLP-76 binding differentially regulates supramolecular activation cluster (SMAC) formation relative to T cell-APC conjugation.
The cytomegalovirus m155 gene product subverts natural killer cell antiviral protection by disruption of H60-NKG2D interactions. Read more about The cytomegalovirus m155 gene product subverts natural killer cell antiviral protection by disruption of H60-NKG2D interactions.
