TBX3 functions as a tumor suppressor downstream of activated CTNNB1 mutants during hepatocarcinogenesis. Read more about TBX3 functions as a tumor suppressor downstream of activated CTNNB1 mutants during hepatocarcinogenesis.
A novel waitlist dropout score for hepatocellular carcinoma - identifying a threshold that predicts worse post-transplant survival. Read more about A novel waitlist dropout score for hepatocellular carcinoma - identifying a threshold that predicts worse post-transplant survival.
MicroRNA-342-3p is a potent tumour suppressor in hepatocellular carcinoma. Read more about MicroRNA-342-3p is a potent tumour suppressor in hepatocellular carcinoma.
Systemic therapies for intrahepatic cholangiocarcinoma. Read more about Systemic therapies for intrahepatic cholangiocarcinoma.
Loss of Fbxw7 synergizes with activated Akt signaling to promote c-Myc dependent cholangiocarcinogenesis. Read more about Loss of Fbxw7 synergizes with activated Akt signaling to promote c-Myc dependent cholangiocarcinogenesis.
Pan-mTOR inhibitor MLN0128 is effective against intrahepatic cholangiocarcinoma in mice. Read more about Pan-mTOR inhibitor MLN0128 is effective against intrahepatic cholangiocarcinoma in mice.
Observational registry of sorafenib use in clinical practice across Child-Pugh subgroups: The GIDEON study. Read more about Observational registry of sorafenib use in clinical practice across Child-Pugh subgroups: The GIDEON study.
Mutations in DCDC2 (doublecortin domain containing protein 2) in neonatal sclerosing cholangitis. Read more about Mutations in DCDC2 (doublecortin domain containing protein 2) in neonatal sclerosing cholangitis.
Inactivation of fatty acid synthase impairs hepatocarcinogenesis driven by AKT in mice and humans. Read more about Inactivation of fatty acid synthase impairs hepatocarcinogenesis driven by AKT in mice and humans.
Essential roles of FoxM1 in Ras-induced liver cancer progression and in cancer cells with stem cell features. Read more about Essential roles of FoxM1 in Ras-induced liver cancer progression and in cancer cells with stem cell features.
