Versatile in vivo regulation of tumor phenotypes by dCas9-mediated transcriptional perturbation. Read more about Versatile in vivo regulation of tumor phenotypes by dCas9-mediated transcriptional perturbation.
The neurofibromin recruitment factor Spred1 binds to the GAP related domain without affecting Ras inactivation. Read more about The neurofibromin recruitment factor Spred1 binds to the GAP related domain without affecting Ras inactivation.
Transmembrane channel-like (tmc) gene regulates Drosophila larval locomotion. Read more about Transmembrane channel-like (tmc) gene regulates Drosophila larval locomotion.
A scalable strategy for high-throughput GFP tagging of endogenous human proteins. Read more about A scalable strategy for high-throughput GFP tagging of endogenous human proteins.
Mechanism of inhibition of human glucose transporter GLUT1 is conserved between cytochalasin B and phenylalanine amides. Read more about Mechanism of inhibition of human glucose transporter GLUT1 is conserved between cytochalasin B and phenylalanine amides.
Ang-2/VEGF bispecific antibody reprograms macrophages and resident microglia to anti-tumor phenotype and prolongs glioblastoma survival. Read more about Ang-2/VEGF bispecific antibody reprograms macrophages and resident microglia to anti-tumor phenotype and prolongs glioblastoma survival.
Quantitative MS-based enzymology of caspases reveals distinct protein substrate specificities, hierarchies, and cellular roles. Read more about Quantitative MS-based enzymology of caspases reveals distinct protein substrate specificities, hierarchies, and cellular roles.
Quantitative proteomic analyses of mammary organoids reveals distinct signatures after exposure to environmental chemicals. Read more about Quantitative proteomic analyses of mammary organoids reveals distinct signatures after exposure to environmental chemicals.
ER stress stimulates production of the key antimicrobial peptide, cathelicidin, by forming a previously unidentified intracellular S1P signaling complex. Read more about ER stress stimulates production of the key antimicrobial peptide, cathelicidin, by forming a previously unidentified intracellular S1P signaling complex.
Transcription-associated processes cause DNA double-strand breaks and translocations in neural stem/progenitor cells. Read more about Transcription-associated processes cause DNA double-strand breaks and translocations in neural stem/progenitor cells.
