CD94 is essential for NK cell-mediated resistance to a lethal viral disease. Read more about CD94 is essential for NK cell-mediated resistance to a lethal viral disease.
Paired immunoglobin-like receptor-B regulates the suppressive function and fate of myeloid-derived suppressor cells. Read more about Paired immunoglobin-like receptor-B regulates the suppressive function and fate of myeloid-derived suppressor cells.
Selective utilization of Toll-like receptor and MyD88 signaling in B cells for enhancement of the antiviral germinal center response. Read more about Selective utilization of Toll-like receptor and MyD88 signaling in B cells for enhancement of the antiviral germinal center response.
Posttranscriptional silencing of effector cytokine mRNA underlies the anergic phenotype of self-reactive T cells. Read more about Posttranscriptional silencing of effector cytokine mRNA underlies the anergic phenotype of self-reactive T cells.
Ubiquitin makes its mark on immune regulation. Read more about Ubiquitin makes its mark on immune regulation.
The ubiquitin modifying enzyme A20 restricts B cell survival and prevents autoimmunity. Read more about The ubiquitin modifying enzyme A20 restricts B cell survival and prevents autoimmunity.
CD45-Csk phosphatase-kinase titration uncouples basal and inducible T cell receptor signaling during thymic development. Read more about CD45-Csk phosphatase-kinase titration uncouples basal and inducible T cell receptor signaling during thymic development.
Macrophage- and dendritic-cell-derived interleukin-15 receptor alpha supports homeostasis of distinct CD8+ T cell subsets. Read more about Macrophage- and dendritic-cell-derived interleukin-15 receptor alpha supports homeostasis of distinct CD8+ T cell subsets.
Is antigen specificity of autoreactive T cells the key to islet entry? Read more about Is antigen specificity of autoreactive T cells the key to islet entry?
Loss of the LAT adaptor converts antigen-responsive T cells into pathogenic effectors that function independently of the T cell receptor. Read more about Loss of the LAT adaptor converts antigen-responsive T cells into pathogenic effectors that function independently of the T cell receptor.
