The effect of induction immunosuppression for kidney transplant on the latent HIV reservoir. Read more about The effect of induction immunosuppression for kidney transplant on the latent HIV reservoir.
Glutamine prevents acute kidney injury by modulating oxidative stress and apoptosis in tubular epithelial cells. Read more about Glutamine prevents acute kidney injury by modulating oxidative stress and apoptosis in tubular epithelial cells.
Immunopathogenesis of hidradenitis suppurativa and response to anti-TNF-α therapy. Read more about Immunopathogenesis of hidradenitis suppurativa and response to anti-TNF-α therapy.
Markers of fungal translocation are elevated during post-acute sequelae of SARS-CoV-2 and induce NF-κB signaling. Read more about Markers of fungal translocation are elevated during post-acute sequelae of SARS-CoV-2 and induce NF-κB signaling.
Immune cells and their inflammatory mediators modify β cells and cause checkpoint inhibitor-induced diabetes. Read more about Immune cells and their inflammatory mediators modify β cells and cause checkpoint inhibitor-induced diabetes.
Markers of fungal translocation are elevated during post-acute sequelae of SARS-CoV-2 and induce NF-κB signaling. Read more about Markers of fungal translocation are elevated during post-acute sequelae of SARS-CoV-2 and induce NF-κB signaling.
IL-13-programmed airway tuft cells produce PGE2, which promotes CFTR-dependent mucociliary function. Read more about IL-13-programmed airway tuft cells produce PGE2, which promotes CFTR-dependent mucociliary function.
β Cell-specific deletion of Zfp148 improves nutrient-stimulated β cell Ca2+ responses. Read more about β Cell-specific deletion of Zfp148 improves nutrient-stimulated β cell Ca2+ responses.
Role of antibodies, inflammatory markers, and echocardiographic findings in postacute cardiopulmonary symptoms after SARS-CoV-2 infection. Read more about Role of antibodies, inflammatory markers, and echocardiographic findings in postacute cardiopulmonary symptoms after SARS-CoV-2 infection.
WEE1 kinase is a therapeutic vulnerability in CIC-DUX4 undifferentiated sarcoma. Read more about WEE1 kinase is a therapeutic vulnerability in CIC-DUX4 undifferentiated sarcoma.
