Human LAT mutation results in immune deficiency and autoimmunity but also raises questions about signaling pathways. Read more about Human LAT mutation results in immune deficiency and autoimmunity but also raises questions about signaling pathways.
Ubiquitin-mediated fluctuations in MHC class II facilitate efficient germinal center B cell responses. Read more about Ubiquitin-mediated fluctuations in MHC class II facilitate efficient germinal center B cell responses.
A novel human autoimmune syndrome caused by combined hypomorphic and activating mutations in ZAP-70. Read more about A novel human autoimmune syndrome caused by combined hypomorphic and activating mutations in ZAP-70.
Type I IFN promotes NK cell expansion during viral infection by protecting NK cells against fratricide. Read more about Type I IFN promotes NK cell expansion during viral infection by protecting NK cells against fratricide.
The G protein-coupled receptor P2RY8 and follicular dendritic cells promote germinal center confinement of B cells, whereas S1PR3 can contribute to their dissemination. Read more about The G protein-coupled receptor P2RY8 and follicular dendritic cells promote germinal center confinement of B cells, whereas S1PR3 can contribute to their dissemination.
Getting close to the action elicits better memories. Read more about Getting close to the action elicits better memories.
The lysophosphatidylserine receptor GPR174 constrains regulatory T cell development and function. Read more about The lysophosphatidylserine receptor GPR174 constrains regulatory T cell development and function.
IgE-activated basophils regulate eosinophil tissue entry by modulating endothelial function. Read more about IgE-activated basophils regulate eosinophil tissue entry by modulating endothelial function.
Cytomegalovirus generates long-lived antigen-specific NK cells with diminished bystander activation to heterologous infection. Read more about Cytomegalovirus generates long-lived antigen-specific NK cells with diminished bystander activation to heterologous infection.