CXCR4 and a cell-extrinsic mechanism control immature B lymphocyte egress from bone marrow. Read more about CXCR4 and a cell-extrinsic mechanism control immature B lymphocyte egress from bone marrow.
Antigen-specific expansion and differentiation of natural killer cells by alloantigen stimulation. Read more about Antigen-specific expansion and differentiation of natural killer cells by alloantigen stimulation.
GPR18 is required for a normal CD8αα intestinal intraepithelial lymphocyte compartment. Read more about GPR18 is required for a normal CD8αα intestinal intraepithelial lymphocyte compartment.
The Src family kinases Hck, Fgr, and Lyn are critical for the generation of the in vivo inflammatory environment without a direct role in leukocyte recruitment. Read more about The Src family kinases Hck, Fgr, and Lyn are critical for the generation of the in vivo inflammatory environment without a direct role in leukocyte recruitment.
Proapoptotic Bim regulates antigen-specific NK cell contraction and the generation of the memory NK cell pool after cytomegalovirus infection. Read more about Proapoptotic Bim regulates antigen-specific NK cell contraction and the generation of the memory NK cell pool after cytomegalovirus infection.
Sphingosine-1-phosphate receptor 2 is critical for follicular helper T cell retention in germinal centers. Read more about Sphingosine-1-phosphate receptor 2 is critical for follicular helper T cell retention in germinal centers.
Leukotriene B4 amplifies eosinophil accumulation in response to nematodes. Read more about Leukotriene B4 amplifies eosinophil accumulation in response to nematodes.
Enhancement of an anti-tumor immune response by transient blockade of central T cell tolerance. Read more about Enhancement of an anti-tumor immune response by transient blockade of central T cell tolerance.
MyD88-dependent interplay between myeloid and endothelial cells in the initiation and progression of obesity-associated inflammatory diseases. Read more about MyD88-dependent interplay between myeloid and endothelial cells in the initiation and progression of obesity-associated inflammatory diseases.
Pten loss in the bone marrow leads to G-CSF-mediated HSC mobilization. Read more about Pten loss in the bone marrow leads to G-CSF-mediated HSC mobilization.
