Dual pressure from antiretroviral therapy and cell-mediated immune response on the human immunodeficiency virus type 1 protease gene. Read more about Dual pressure from antiretroviral therapy and cell-mediated immune response on the human immunodeficiency virus type 1 protease gene.
Developing novel oncolytic adenoviruses through bioselection. Read more about Developing novel oncolytic adenoviruses through bioselection.
Increased virus replication and virulence after serial passage of human immunodeficiency virus type 2 in baboons. Read more about Increased virus replication and virulence after serial passage of human immunodeficiency virus type 2 in baboons.
Tissue-specific transcriptional targeting of a replication-competent retroviral vector. Read more about Tissue-specific transcriptional targeting of a replication-competent retroviral vector.
Evolution of phenotypic drug susceptibility and viral replication capacity during long-term virologic failure of protease inhibitor therapy in human immunodeficiency virus-infected adults. Read more about Evolution of phenotypic drug susceptibility and viral replication capacity during long-term virologic failure of protease inhibitor therapy in human immunodeficiency virus-infected adults.
Integrin alphavbeta1 is a receptor for foot-and-mouth disease virus. Read more about Integrin alphavbeta1 is a receptor for foot-and-mouth disease virus.
Altered substrate specificity of drug-resistant human immunodeficiency virus type 1 protease. Read more about Altered substrate specificity of drug-resistant human immunodeficiency virus type 1 protease.
Vesicular stomatitis virus G-pseudotyped lentivirus vectors mediate efficient apical transduction of polarized quiescent primary alveolar epithelial cells. Read more about Vesicular stomatitis virus G-pseudotyped lentivirus vectors mediate efficient apical transduction of polarized quiescent primary alveolar epithelial cells.
The pathogenicity of human immunodeficiency virus (HIV) type 1 Nef in CD4C/HIV transgenic mice is abolished by mutation of its SH3-binding domain, and disease development is delayed in the absence of Hck. Read more about The pathogenicity of human immunodeficiency virus (HIV) type 1 Nef in CD4C/HIV transgenic mice is abolished by mutation of its SH3-binding domain, and disease development is delayed in the absence of Hck.
Retroviral integration at the Epi1 locus cooperates with Nf1 gene loss in the progression to acute myeloid leukemia. Read more about Retroviral integration at the Epi1 locus cooperates with Nf1 gene loss in the progression to acute myeloid leukemia.
